Search results for "Inbred F344"

showing 10 items of 30 documents

Proliferative activity and tumorigenic conversion: impact on cellular metabolism in 3-D culture

2001

Oxygen consumption, glucose, lactate, and ATP concentrations, as well as glucose and lactate turnover rates, have been studied in a three-dimensional carcinogenesis model of differently transformed rat embryo fibroblasts (spontaneously immortalized Rat1 and myc-transfected M1, and the ras-transfected, tumorigenic descendants Rat1-T1 and MR1) to determine metabolic alterations that accompany tumorigenic conversion. Various bioluminescence techniques, thymidine labeling, measurement of[Formula: see text] distributions with microelectrodes, and determination of cellular oxygen uptake rates (Q˙[Formula: see text]) have been applied. In the ras-transfected, tumorigenic spheroid types, the size d…

Cell divisionPhysiologyBiologymedicine.disease_causeDiffusionchemistry.chemical_compoundAdenosine TriphosphateOxygen ConsumptionSpheroids CellularmedicineAnimalsLactic AcidFibroblastCell Line TransformedCell growthCell BiologyTransfectionFibroblastsEmbryo MammalianRats Inbred F344In vitroRatsLactic acidOxygenCell Transformation NeoplasticGlucosemedicine.anatomical_structurechemistryBiochemistryembryonic structuresCarcinogenesisAdenosine triphosphateCell DivisionAmerican Journal of Physiology-Cell Physiology
researchProduct

NMDA receptor antagonist treatment increases the production of new neurons in the aged rat hippocampus

2002

The production of new neurons declines during adulthood and persists, although at very low levels, in the aged hippocampus. Since neurogenesis in young adults has been related to learning and memory, its reduction may contribute to the age-related impairments in these abilities. Adrenalectomy (ADX) enhances neurogenesis in the aged hippocampus, although it also induces neuronal cell death. Since the administration of an NMDA receptor antagonist enhances neurogenesis in young adult rats without deleterious morphological effects, we have tested whether neurogenesis could be reactivated in aged rats. Our study shows that cell proliferation, cell death, neurogenesis and the number of radial gli…

Doublecortin Domain ProteinsAgingmedicine.medical_specialtyAntimetabolitesCell SurvivalCentral nervous systemHippocampusNerve Tissue ProteinsBiologyReceptors N-Methyl-D-AspartateInternal medicinemedicineAnimalsNeuronsCell DeathGeneral NeuroscienceNeuropeptidesNeurogenesisGlutamate receptorAntagonistAdrenalectomyNestinImmunohistochemistryRats Inbred F344RatsDoublecortinEndocrinologymedicine.anatomical_structure2-Amino-5-phosphonovalerateBromodeoxyuridinenervous systemDentate Gyrusbiology.proteinNMDA receptorFemaleNeurology (clinical)Geriatrics and GerontologyExcitatory Amino Acid AntagonistsMicrotubule-Associated ProteinsNeurogliaBiomarkersCell DivisionDevelopmental BiologyNeurobiology of Aging
researchProduct

Comparison of enzyme phenotypes in human bladder tumours and experimentally induced hyperplastic and neoplastic lesions of the rat urinary bladder. A…

1988

The expression of a number of enzymes involved in drug metabolism, membrane function etc. was compared in hyperplastic and neoplastic lesions of the rat bladder and in human bladder tumours. Transitional cell carcinomas (TCC) in both rat and Man were characterized by decreased alkaline phosphatase (ALP) and increased gamma-glutamyl transpeptidase (GGT), beta-glucuronidase (beta-G1), succinate dehydrogenase (SD) and glucose-6-phosphate dehydrogenase (G6PD) activities. In addition, binding for antibodies specific for different cytochrome P-450 species (UT50, PB3a, MC1, MC2) and microsomal epoxide hydrolase (mEHb) was elevated in both murine and human tumours. Comparison of the enzyme phenotyp…

Malemedicine.medical_specialtyPathologyUrinary BladderGlucosephosphate DehydrogenaseInternal medicinemedicineAnimalsHumansNeoplastic transformationEnzyme inducerGlucuronidaseCarcinoma Transitional CellUrinary bladderHyperplasiabiologyHistocytochemistrySuccinate dehydrogenasegamma-GlutamyltransferaseHyperplasiamedicine.diseaseAlkaline PhosphataseImmunohistochemistryRats Inbred F344RatsSuccinate DehydrogenaseEndocrinologymedicine.anatomical_structurePhenotypeUrinary Bladder NeoplasmsMicrosomal epoxide hydrolasebiology.proteinImmunohistochemistryDrug metabolismVirchows Archiv. B, Cell pathology including molecular pathology
researchProduct

Liver regeneration induced by a designer human IL‐6/ sIL‐6R fusion protein reverses severe hepatocellular injury

2000

The cytokine IL-6 plays a significant role in liver regeneration in conjunction with additional growth factors (HGF, TNF-α, and TGF-α). Many IL-6 effects depend on a naturally occurring soluble IL-6 receptor (sIL-6R). Here, the chimeric protein hyper-IL-6, constructed from the human IL-6 protein fused to a truncated form of its receptor, was found to have superagonistic IL-6 properties, and as such, enhanced liver cell regeneration. Hyper-IL-6 reversed the state of hepatotoxicity and enhanced the survival rates of rats suffering from fulminant hepatic failure after D-galactosamine administration. The hyper-IL-6 protein has a significant potential for use in the treatment of severe human liv…

MalePathologymedicine.medical_specialtyRecombinant Fusion Proteinsmedicine.medical_treatmentApoptosisGalactosamineThioacetamideBiochemistryFulminant hepatic failureGeneticsmedicineAnimalsHumansReceptorInterleukin 6Molecular BiologybiologyInterleukin-6ChemistryLiver cellRegeneration (biology)Receptors InterleukinReceptors Interleukin-6Fusion proteinRats Inbred F344Liver regenerationLiver RegenerationRatsDisease Models AnimalCytokineCancer researchbiology.proteinCell DivisionLiver FailureBiotechnologyThe FASEB Journal
researchProduct

Time-dependence and differential induction of rat and guinea pig peroxisomal beta-oxidation, palmitoyl-CoA hydrolase, cytosolic and microsomal epoxid…

1988

Fischer-344 rats and Hartley guinea pigs received a diet containing 0.01% (w/w), 0.05% (w/w), or 0.25% (w/w) of the hypolipidemic drug fenofibrate. Rats were treated for 4, 7, 14, or 21 days, and a clear dose-dependent and weak time-dependent increase in liver/body weight ratio was observed. The specific activity of peroxisomal beta-oxidation increased linearly with time at all concentrations used. A dose-dependent increase in cEH was observed, but the activity remained constant after treatment for 7 days. Enhancement of palmitoyl-CoA hydrolase was dose-dependent, but was similar at all 4 time points investigated. In contrast to the other enzyme activities, mEH was not or only minimally (le…

MaleCancer Researchmedicine.medical_specialtyTime FactorsTiadenolGuinea PigsBiologyMicrobodiesGuinea pigCytosolInternal medicineMicrosomesHydrolasemedicineAnimalsHypolipidemic Agentschemistry.chemical_classificationEpoxide HydrolasesClofibrateFenofibrateGeneral MedicineRats Inbred F344RatsPalmitoyl-CoA hydrolaseEnzymeEndocrinologyOncologychemistryBiochemistryPalmitoyl-CoA HydrolaseMicrosomal epoxide hydrolaseEnzyme InductionThiolester Hydrolasesmedicine.drugJournal of cancer research and clinical oncology
researchProduct

Adult rat myelin enhances axonal outgrowth from neural stem cells.

2018

Axon regeneration after spinal cord injury (SCI) is attenuated by growth inhibitory molecules associated with myelin. We report that rat myelin stimulated the growth of axons emerging from rat neural progenitor cells (NPCs) transplanted into sites of SCI in adult rat recipients. When plated on a myelin substrate, neurite outgrowth from rat NPCs and from human induced pluripotent stem cell (iPSC)-derived neural stem cells (NSCs) was enhanced threefold. In vivo, rat NPCs and human iPSC-derived NSCs extended greater numbers of axons through adult central nervous system white matter than through gray matter and preferentially associated with rat host myelin. Mechanistic investigations excluded …

0301 basic medicineAgingNeuronalNudeMessengerNeurodegenerativeInbred C57BLRegenerative MedicineMedical and Health SciencesMyelinMiceNeural Stem CellsStem Cell Research - Nonembryonic - HumanCyclic AMPAxonPhosphorylationGray MatterInduced pluripotent stem cellExtracellular Signal-Regulated MAP KinasesSpinal Cord InjuryMyelin SheathInbred F344Neuronal growth regulator 1Stem Cell Research - Induced Pluripotent Stem Cell - HumanChemistryGeneral MedicineBiological SciencesWhite MatterNeural stem cellCell biologymedicine.anatomical_structureSpinal Cord5.1 PharmaceuticalsNeurologicalFemaleStem Cell Research - Nonembryonic - Non-HumanDevelopment of treatments and therapeutic interventionsPhysical Injury - Accidents and Adverse EffectsNeuriteCell Adhesion Molecules NeuronalCentral nervous systemNeuronal OutgrowthArticleWhite matter03 medical and health sciencesRats NudemedicineAnimalsHumansRNA MessengerStem Cell Research - Embryonic - HumanTraumatic Head and Spine InjuryTransplantationStem Cell Research - Induced Pluripotent Stem CellNeurosciencesStem Cell ResearchRats Inbred F344AxonsRatsMice Inbred C57BL030104 developmental biologynervous systemChondroitin Sulfate ProteoglycansRNACell Adhesion Molecules
researchProduct

Signaling through BMPR-IA regulates quiescence and long-term activity of neural stem cells in the adult hippocampus.

2010

SummaryNeural stem cells (NSCs) in the adult hippocampus divide infrequently, and the molecules that modulate their quiescence are largely unknown. Here, we show that bone morphogenetic protein (BMP) signaling is active in hippocampal NSCs, downstream of BMPR-IA. BMPs reversibly diminish proliferation of cultured NSCs while maintaining their undifferentiated state. In vivo, acute blockade of BMP signaling in the hippocampus by intracerebral infusion of Noggin first recruits quiescent NSCs into the cycle and increases neurogenesis; subsequently, it leads to decreased stem cell division and depletion of precursors and newborn neurons. Consistently, selective ablation of Bmpr1a in hippocampal …

medicine.medical_specialtyanimal structuresGenetic VectorsHippocampal formationBiologyBone morphogenetic proteinHippocampusModels BiologicalMOLNEUROCell LineMiceNeural Stem CellsInternal medicineGeneticsmedicineAnimalsHumansNogginBone Morphogenetic Protein Receptors Type ICells Culturedreproductive and urinary physiologySmad4 ProteinNeuronsReverse Transcriptase Polymerase Chain ReactionStem CellsCell CycleLentivirusNeurogenesisCentral-nervous-system; Bone morphogenetic protein; Dentate gyrus; Progenitor cells; Neurogenesis; Expression; Receptor; Noggin; Brain; DifferentiationCell BiologyFlow CytometrySTEMCELLRats Inbred F344BMPR1ANeural stem cellRatsCell biologyEndocrinologyStem cell divisionnervous systemembryonic structuresMolecular MedicineStem cellbiological phenomena cell phenomena and immunityCarrier ProteinsSignal Transduction
researchProduct

Efficient, non-toxic anion transport by synthetic carriers in cells and epithelia.

2016

Transmembrane anion transporters (anionophores) have potential for new modes of biological activity, including therapeutic applications. In particular they might replace the activity of defective anion channels in conditions such as cystic fibrosis. However, data on the biological effects of anionophores are scarce, and it remains uncertain whether such molecules are fundamentally toxic. Here, we report a biological study of an extensive series of powerful anion carriers. Fifteen anionophores were assayed in single cells by monitoring anion transport in real time through fluorescence emission from halide-sensitive yellow fluorescent protein. A bis-(p-nitrophenyl)ureidodecalin shows especial…

Yellow fluorescent proteinpotencyGeneral Chemical Engineeringsynthetic anion carriersCystic Fibrosis Transmembrane Conductance Regulator01 natural sciencesMadin Darby Canine Kidney CellsCell membranedeliverabilityta116Drug CarriersbiologyMolecular StructureChemistryBiological activitypersistenceCystic fibrosis transmembrane conductance regulatorTransmembrane proteinanionophoresmedicine.anatomical_structureBiochemistryPhosphatidylcholinesSteroidsChlorineAnionsCell SurvivalNaphthalenesta3111010402 general chemistryDogsBacterial ProteinsCyclohexanesmedicineAnimalsHumansIon transporterCell ProliferationIon Transport010405 organic chemistryCell MembranetoxicityTransporterEpithelial CellsHydrogen BondingGeneral ChemistryRats Inbred F3440104 chemical sciencesElectrophysiological PhenomenaLuminescent ProteinsMicroscopy FluorescenceCell cultureDrug Designbiology.proteinHeLa CellsNature chemistry
researchProduct

Tissue gradients of energy metabolites mirror oxygen tension gradients in a rat mammary carcinoma model

2001

Abstract Purpose: It has been shown that oxygen gradients exist in R3230AC tumors grown in window chambers. The fascial surface is better oxygenated than the tumor surface. The purpose of the present study was to determine whether gradients exist for energy metabolites and other end points related to oxygen transport. Methods and Materials: Imaging bioluminescence was used to measure ATP, glucose, and lactate in cryosections of R3230AC tumors. Mean vessel density and hypoxic tissue fraction were assessed using immunohistochemistry. Tumor redox ratio was assessed by redox ratio scanning. Results: Lactate content and hypoxic fraction increased, whereas ATP, glucose, redox ratio, and vessel de…

Cancer ResearchPathologymedicine.medical_specialtychemistry.chemical_elementOxygenMicrocirculationAdenosine TriphosphatemedicineAnimalsPimonidazoleRadiology Nuclear Medicine and imagingLactic AcidRadiationTumor hypoxiabusiness.industryMicrocirculationOxygen transportMammary Neoplasms ExperimentalRadiobiologyOxygenationMetabolismCell HypoxiaRats Inbred F344RatsOxygen tensionOxygenGlucoseOncologychemistryLuminescent MeasurementsBiophysicsDiffusion Chambers CulturebusinessOxidation-ReductionInternational Journal of Radiation Oncology*Biology*Physics
researchProduct

In vivo formation of aflatoxin B1-DNA adducts in parenchymal and non-parenchymal cells of rat liver

1992

The induction of hepatocellular carcinoma from liver parenchymal cells in laboratory animals by aflatoxin B1 (AFB1) is well documented. In contrast no tumours arising from the sinusoidal cell population have been reported after exposure to AFB1. The apparent resistance of the latter cell type was investigated at the level of DNA adduct formation in vivo in male Sprague-Dawley rats. Liver parenchymal and non-parenchymal cell populations were isolated from rats at 20 min and 1, 24 and 72 h after administration of 240 microCi (0.6 mg) [G-3H]AFB1/kg. AFB1-DNA binding was observed in both liver cell subpopulations and was 3- to 5-fold higher in parenchymal cells than in non-parenchymal cells. Th…

MaleCancer ResearchCell typePathologymedicine.medical_specialtyAflatoxin B1Time FactorsPopulationCellBiologyDNA AdductsIn vivoDNA adductmedicineAnimalseducationeducation.field_of_studyLiver cellRats Inbred StrainsDNAGeneral Medicinemedicine.diseaseRats Inbred F344Ratsmedicine.anatomical_structureLiverHepatocyteHepatocellular carcinomaCarcinogenesis
researchProduct